Immunodeficiency syndrome  acquired immunodeficiency virus by the (human immunodeficiency vius-HIV) was first described in 1981 and became a global epidemic

Eye injury in AIDS was first announced in 1982. Eye manifestation in AIDS in many forms, occurs in most parts of the eye and usually occurrs in late stages of disease but also when the first signs of help towards the discovery of HIV. Damage to eyes is one common manifestation of HIV infection, 40-70% of AIDS patients with eye injury.

Eye injury AIDS is divided into four groups: HIV retinopathy, opportunistic infections caused by virus, bacteria, fungi, parasites, eye nerve injury, and organize new products (less common)

HIV Retinopathy

Circuit retinal disease by HIV is the most common eye injury in patients with AIDS, representing about 50% of HIV cases last stage. HIV Retinopathy is damage to the retina microchip no infection, characterized by sports produce more cotton, for aneurysm, retinal hemorrhages, dilated capillaries and the capillary zone rules.

Produce more cotton in 50% of patients with HIV disease progression, the earliest signs and almost constant in HIV retinopathy manilifested locally, infaction of nerve fibers in the layers of the retina. Average time spendd to produce more cotton is 6-12 weeks.

Retinal hemorrhages or dot form is less common form candles produce more cotton, 20% in HIV disease progression. Retinal hemorrhages spend a few weeks.

There maybe other variations, such as microhip capillary relaxing with aneurysm of the retinal capillaries or the switch circuit is detected by fluorescence capture.

Opportunistic infections

There are many dark retinal infections in HIV patients. These oppotunistic infections common in dark retina in patients with HIV infection, including viral resistance retinal cells, retinal necrosis caused by herpes  virus or shingles level, retinal inflammation y black workers, black retinal inflammation due to Toxoplasma, inflammation of the retina caused by black fungi.

Acute retinal cells by the virus resistance

Acute retinal cell length viral (cytomegalovirus-CMV) severe in people with HIV, opportinistic infection are most common in eye syndrome in patients with AIDS.

Patients with blurred vision, can see the aircrat can hover like flies or flasing light ahead.

Jute eue, inflammation of the retina with two forms of expression:

Acute retinal fibrous scaring degradation development speckled pigmentation

Inflammation around the blood vessels when the retina is like the dew tree damage is less common.

Complications retinal detachment occurs in 15-30% of cases, espeically when patients with long tear in the retinal necrosis.

Test: CMV antibody in the blood and eyeballs in service, be buired in the cell nucleus and viral swabs cultured rentinal biopsy

Treatment of acute rential viral tools include the treatment of active inflammatory retinal complications and treatment of rentinal detachment. Treatment os acute rentinal activity with antiviral drugs resistance cells (Ganciclovir, Valgancirclovir, Foscarnet, Cidoclovir) to treat HIV infection and increase immunity. Treatment  of rentinal detachment surgery with glass service combined silicone oil pump internal label.

Retinal necrosis-induced herpes virus, shingles

Acute retinal necrosis caused by herpes zoster or herpes simplex virus types 1 and 2 causes. Injury in both eyes simultaneously.

Reduced vision, patients can see the files hovering like flying ahead.

Jute seen drivers retial necrosis in the white editors. Inflammation often code rentinal aterioles and rentinal hermorrhage. When the inflammatory degradation development, retinal necrosis drivers replaced by scarring and pigmentation retinal atrophy.

Acute retinal necrosis or associated with the shingles skin and damage the central nervous system.

Test: a herpes virus antibodies in the blood and water service, detection of herpes virus DNA in swabs glass cutting services.

Common complications are retinal detachment due to holes, tears in retinal necrosis and retinal proliferation glass service. Retinal detachment occurred in 3 months after disease onset, and met in 2/3 patients.

Treatment of acute retinal necrosis caused by herpes virus, including antiviral drugs (Acyclovir), anti-inflammatory (corticosteroids only be used after 1-2 days using anti-virus to avoid the mulptiplication of the virus), anti-thrombotic (aspirin), reserves, retinal detechment (after the laser treatment of rentinal necrosis) and treatment of retinal detechment (laser cutting services combining glass and silicone oil internal label). HIV treatment to enhance immunity.

Retinal inflammation y black workers

Acute retinal inflammation caused by tuberculosis is usually spread from dark desert on the retina, or in the form of labor statistics.

Acute retinal black workers expressed by older workers as employees or tumor. They maybe one or more older wokers, often in extreme trauma later.

Common retinal cascular inflammation, usually into a vein inflammation. Acute blood vessels can cause swithches to rentinal vascular complications of new chemic retina

Chest x-ray may show pulmonary TB, positive Mantoux reaction

Need medical consultation with a TB specialist, used to coordinate anti-TB drugs according to whether the 6-9 months (group B vitamins sould be included to limit the complications of neurological poisoning due to market anti-TB drug ethambuto), combined HIV rteatment to increase immunity.

Acute retinal tar by Toxolasma

Vision service decreased by opaque glass or injury to the imperial intrusion point. Patients can see the flies hovering flight as translated by the glass front is cloudy, there is the veiw transformaion.

Typical case of acute retinal drives often cream-colored in the exteme, size 0.1-2mm, retinal edema, localized inflammation glass service before retinal inflammatory drive to the image of  “hadlights in the fof”. There maybe one more drives in the second eye inflammation. Usually drive inflammation in the retinal blood vessels next to prove the agent to spread by blood, not re-inflammation in the retinal blood vessels next to prove th agent to spread by blood, not re-inflammation activity. And may include meningitis Portugal before, rentinal vascular inflammation. Drive a few months after retinal scars shrik inflamed retinal pigmented black.

In AIDS patients, Toxoplasma eye injury due to trauma often associated with central nervous system should need brain scans to detect brain damage by Toxoplasma.

IgG and IgM antibody anti Toxoplasma gondil in serum and in civil service label. Effecive price eye ranslated antibodies in the serum higher. Igm positive signs of new infection.

Treatment with anti-Toxoplasma as pyrimethamin, sulfadiazin, clindamyxin, but not with corticold atovaquon.

Acute retinal dark fungal

Acute retinal fungal infections are associated with dark desert. Candida and fungas Aspergillus is the most common cause of acute retinal tar endogenous fungal.